The GIHG was created by the vision of internationally recognized researchers and it aspires to enrich the scientific environment of Greece and to become a world leader in research on genetic diversity and its connection with various monogenic and multifactorial diseases.
Professor Christos (Kitsos) Louis, IMBB researcher, was honored for his significant contributions to the understanding of the biology of the malaria mosquito on July 27 at the Mosquito Kolymbari Conference 2022, held at the Orthodox Academy of Crete.
Establishment of the Hellenic Network of Molecular Oncology, a Pan-Hellenic hub in the field of oncology
In nature, there is a constant evolutionary battle between microbes and their hosts, during which, many bacteria secrete specific proteins into the host’s cells, aiming to alter its physiology, while increasing pestiferous activity.
A team of scientists from the University of Minnesota Medical School, University of Texas Health San Antonio, and the Biomedical Research Institute of FORTH, found that the disruption of a circadian clock gene may be involved in the development of autism spectrum disorder.
FORTH researchers reveal how metabolic reprogramming in mitochondria promotes or undermines survival and longevity
The new findings of the study are published today in the premier international scientific journal Nature Communications.
How does nature utilize the common repertoire of folds in proteins to differentiate their specificity, and ultimately their function? This long-standing and fundamental question was addressed in a study conducted at the Institute of Molecular Biology and Biotechnology of the Foundation for Research & Technology-Hellas (FORTH-IMBB).
IMBB researchers reveal that DNA damage in tissue-infiltrating macrophages triggers an exosome-based metabolic reprogramming leading to chronic inflammation.
Research carried out at the Institute of Molecular Biology and Biotechnology (IMBB) of FORTH, provides evidence that persistent DNA damage triggers an exosome-based, metabolic reprogramming that leads to chronic inflammation and tissue pathology in DNA repair-deficient progeroid syndromes and likely also during aging.
The findings of the study, published today in the premier international scientific journal Cell Reports, reveal a novel molecular mechanism underlying the detrimental effects of BER imbalance in the pathogenesis of Parkinson’s disease.
Cardiotoxicity, as a side effect of breast cancer treatment, at the heart of a new European research project
The new European research project CARDIOCARE, will focus on the management of the elderly multimorbid patients with breast cancer therapy-induced cardiac toxicity.
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